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Treatment with NAC increased the abundance of NADPH which in turn resulted in increased catabolism by NADPH-dependent kynurenine hydroxylase leading to lower levels of kynurenine which subsequently inhibited the mTOR pathway in those T cells [19]

Cell 178 , 473490 (2019)

(2011) showed that the P2Y1 activation and consequent Ca 2+ -dependent glutamate release in astrocytes from the dentate gyrus also involve the tumor necrosis factor-alpha (TNF), which at physiological concentrations (pM range), favors the adequate exocytosis of astrocytic glutamatergic vesicles