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Moreover, TNF- contributes to endothelial dysfunction by activating the NF-B and mitogen-activated protein kinase (MAPK) signaling pathways, which in turn promote the transcription of additional pro-inflammatory cytokines and apoptosis-related genes
doi: 10.3389/fpsyt.2022.951376
In vitro reaction of 1,2-DCP with GSH, in vitro transport studies using ABCC2-expressing membrane vesicles, and other detailed methods are provided in Supplementary Information
K1 cells appeared to have much higher expression of these proteins compared to MDA-T32, MDA-T68 and TPC-1 cells (Fig
