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The specific issue could be a strong effect on increasing angiogenesis, increased VEGF, increased egr-1 gene, increased NO or eNOS stimulation, and increased free radical formation, interaction with multiple systems, and unescapable risks of unanticipated adverse effects due to pleiotropic effects
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Recent studies of the heart, lungs, and small blood vessels of the kidneys have shown that the EG is involved in some manifestations of endothelial dysfunction in sepsis (53, 55), and Song et al
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