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Abstract Etiological therapies aim at repairing the underlying cause of cystic fibrosis (CF), which is the functional defect of the cystic fibrosis transmembrane conductance regulator (CFTR) protein owing to mutations in the CFTR gene

Non-pathogenic Escherichia coli enhance Stx2a production of E

Instead, they may rely more heavily on cheaper, processed foods that lack essential nutrients

The desmosomeIF network plays a crucial role in balancing the forces of mechanical stress in many systems, allowing excess contractile activity to be offloaded (2)