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Therapeutic implications: T1DM-associated metabolic insultsS1P depletion, glucolipotoxicityconverge on autophagic and epigenetic mechanisms to disable immunoregulatory cells

According to the results of our study, ALCAR prevented the formation and development of cerebral vasospasm

This is due to a reduction in the inflammatory response via downregulation of TNF- and IL-1 expression to nearly normal levels [155]

2024;4:e0003888
