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In a 2001 study published in Endocrinology , Heffernan and colleagues reported that chronic AOD-9604 administration reduced body weight gain, decreased adipose tissue mass, and increased fat oxidation in obese mice without altering IGF-1 levels or glucose metabolism effects that were abolished in beta3 adrenergic receptor (3-AR) knockout mice, implicating 3-AR signaling as the mechanism of action [1]

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Hepatic effets: Elevations of one or more of the liver enzymes may occur with diclofenac treatment

Translating that directly to human research doses is complicated by the obese rodent model problems described earlier and the difference in metabolic rate scaling between species